Current Immunotherapeutic strategies have primarily focused on checkpoint inhibitors targeting conventional T cells. Mucosal-associated invariant T (MAIT) cells represent an abundant innate-like T cell subtype in the human liver 1. MAIT cells are assigned crucial roles in regulating immunity and inflammation, yet their role in liver cancer remains elusive. We have previously profiled MAITs in the context of hepatocellular carcinoma (HCC) using scRNA-seq, flow cytometry, and co-detection by indexing (CODEX) imaging of paired patient samples. These analyses highlight the heterogeneity and dysfunctionality of MAIT cells in HCC and their defective capacity to infiltrate liver tumors. Co-localization in the adjacent liver and interaction between niche-occupying CSF1R+PD-L1+ tumor-associated macrophages (TAMs) and MAIT cells was identified as a key regulatory element of MAIT cell dysfunction. Perturbation of this cell-cell interaction reinvigorated MAIT cell cytotoxicity 2. Activation of MAITs using 5-OP-RU and CpG led to anti-tumor immunity in murine liver cancer models 3. We are currently studying how MAIT cell exert their anti-tumor function in the liver.